Haloperidol

證據等級: L5 預測適應症: 10

目錄

  1. Haloperidol
  2. Haloperidol: From Schizophrenia/Psychotic Disorders to Manic Bipolar Affective Disorder
    1. One-Sentence Summary
    2. Quick Overview
    3. Why is This Prediction Reasonable?
    4. Clinical Trial Evidence
    5. Literature Evidence
    6. South Africa Market Information
    7. Safety Considerations
    8. Conclusion and Next Steps
    9. Disclaimer

## 藥師評估報告

Haloperidol: From Schizophrenia/Psychotic Disorders to Manic Bipolar Affective Disorder

Note on scope: This evidence pack lists 10 TxGNN-predicted indications for haloperidol. Nine of the ten — including the single highest-scoring prediction (“congenital disorder of glycosylation with defective fucosylation”) — have zero supporting clinical trials or literature and are flagged in the source data as Evidence Level L5 / Hold. Only the 10th-ranked prediction, Manic Bipolar Affective Disorder, is backed by real trial and publication evidence (Evidence Level L1). This report focuses on that indication, as it is the only one with clinical substance to evaluate.

One-Sentence Summary

Haloperidol is a first-generation (typical) D2 dopamine receptor antagonist, historically used in psychotic disorders; formal SAHPRA-registered indication text is not available in this evidence pack. The TxGNN model predicts it may be effective for Manic Bipolar Affective Disorder, a prediction that is unusually well-supported for this drug — 9 clinical trials (three of them Phase 3, completed, with haloperidol as an active comparator) and 20 publications are on record.

Quick Overview

Item Content
Original Indication Not available (no SAHPRA licence records in evidence pack); per known pharmacology, haloperidol is a typical antipsychotic used for psychotic disorders
Predicted New Indication Manic Bipolar Affective Disorder
TxGNN Prediction Score 99.83%
Evidence Level L1
South Africa Market Status Not Marketed (per evidence pack)
Number of SAHPRA Registrations 0
Recommended Decision Proceed with Guardrails

Why is This Prediction Reasonable?

Detailed mechanism-of-action data was not available as a structured field (marked as a Data Gap in the evidence pack). However, the evidence pack’s own rationale analysis identifies haloperidol as a classic D2 dopamine receptor antagonist that suppresses excess mesolimbic dopaminergic activity — one of the core pathophysiological mechanisms implicated in acute mania.

Unlike most of the other TxGNN-predicted indications for this drug (which involve rare congenital, ophthalmologic, or neurodevelopmental conditions with no plausible mechanistic link to dopamine antagonism), mania and psychosis share overlapping neurochemical pathways. Haloperidol is already established in real-world psychiatric practice as an active comparator and adjunct in acute manic episodes of bipolar I disorder — this is therefore less a novel repurposing hypothesis and more a consolidation of existing clinical evidence into a formal indication assessment.

Clinical Trial Evidence

Trial Number Phase Status Enrollment Key Findings
NCT00253162 Phase 3 Completed 439 Risperidone vs. placebo vs. haloperidol in manic episodes of bipolar I disorder; haloperidol used as active comparator over 12 weeks
NCT00129220 Phase 3 Completed 224 Placebo- and haloperidol-controlled trial of olanzapine in manic/mixed bipolar I episodes; haloperidol as active comparator
NCT00253149 Phase 3 Completed 158 Risperidone vs. placebo vs. haloperidol as add-on therapy to mood stabilizers in manic bipolar disorder
NCT00097266 Phase 3 Completed 615 Aripiprazole monotherapy vs. placebo in acute mania; haloperidol’s specific role as comparator arm needs confirmation
NCT04327843 Phase 3 Completed 22 Long-acting injectable antipsychotic plus adherence-focused behavioural programme for chronic psychotic disorders (Tanzania); mania included but population/setting is specific
NCT00126009 Phase 2 Completed 120 Valproate-amisulpride vs. valproate-haloperidol in bipolar I manic episode; haloperidol arm not the primary focus of the title
NCT00767715 Phase 4 Terminated 11 Olanzapine vs. conventional antipsychotics (incl. haloperidol) in acute mania (Sweden); terminated early, small sample
NCT03541031 N/A Unknown 120 Micronutrient/fish-oil supplementation as adjunct in bipolar disorder; not directly evaluating haloperidol
NCT06049953 N/A Recruiting 200 Observational study of antenatal antipsychotic exposure and maternal/infant outcomes; not an efficacy trial

Literature Evidence

PMID Year Type Journal Key Findings
22134043 2012 RCT Journal of Affective Disorders Randomized, double-blind, placebo- and haloperidol-controlled trial of olanzapine in Japanese patients with manic/mixed bipolar I episodes
369472 1979 RCT Archives of General Psychiatry Double-blind controlled trial of lithium plus haloperidol vs. placebo plus haloperidol in excited schizoaffective disorder
3312180 1987 RCT Journal of Clinical Psychiatry Double-blind controlled comparison of clonazepam vs. lithium and vs. haloperidol in acute mania
34642461 2022 Review (network meta-analysis) Molecular Psychiatry Systematic review/NMA of RCTs for acute bipolar mania, comparing efficacy, tolerability and safety across agents including haloperidol
33460070 2020 Review Acta Psychiatrica Scandinavica Evidence-based treatment recommendations for acute manic episodes, covering mood stabilizer and antipsychotic choice
22070611 2012 Review CNS Neuroscience & Therapeutics Refractory bipolar disorder review; notes adding haloperidol, risperidone, olanzapine, quetiapine, or aripiprazole to partial responders
10343182 1999 Observational/mechanistic Neuropsychobiology Lithium and haloperidol treatment differentially affect leukocyte Gαs protein levels in bipolar affective disorder
18344731 2008 Systematic review Journal of Clinical Psychopharmacology Antipsychotic-induced extrapyramidal side effects in bipolar disorder and schizophrenia — safety-relevant comparison
39756485 2025 Retrospective cohort Journal of Affective Disorders Real-world analysis of long-acting injectable antipsychotics added during manic episodes and effect on rehospitalization
36789916 2023 Review BMJ Mental Health Comparison of antipsychotic dose equivalents between acute mania and schizophrenia

South Africa Market Information

The evidence pack shows 0 SAHPRA product registrations and a market status of “Not Marketed” for haloperidol. This is unusual for a long-established, widely used generic antipsychotic and should be treated as a data gap rather than confirmed absence from the South African market — it needs direct verification against the current SAHPRA product register before this factors into any decision.

Safety Considerations

Please refer to the SAHPRA-approved Professional Information (PI) for safety information. Report adverse drug reactions to SAHPRA.

(Key warnings, contraindications, and drug-interaction data were all returned as data gaps or “not found” in this evidence pack — this is logged as a Blocking gap (DG001) that must be closed before any safety assessment.)

Conclusion and Next Steps

Decision: Proceed with Guardrails

Rationale: Three completed Phase 3 RCTs use haloperidol as an active comparator in acute manic episodes of bipolar I disorder, consistent with its established D2-antagonist mechanism — this is corroborating existing practice more than a speculative new hypothesis. However, the complete absence of SAHPRA safety/labelling data and an apparently contradictory “not marketed” status mean this cannot move to Go without closing those gaps.

To proceed, the following is needed:

  • SAHPRA Professional Information (PI): key warnings, contraindications (currently Blocking gap DG001)
  • Confirmation of haloperidol’s actual SAHPRA registration/market status in South Africa (data shows 0 licences, which needs verification)
  • Formal mechanism-of-action documentation from DrugBank (currently High-severity gap DG002)
  • A re-run drug-drug interaction query (current query returned “not_found”)

    Disclaimer

This content is for research purposes only and does not constitute medical advice. Clinical validation is required before any clinical application.



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